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Our study provides a novel peptide drug designed to eliminate senescent cancer cells based on the structural information of the FOXO4-p53 interaction
This CXCR4-CXCL12 signaling pathway activates Caspase-3, leading to apoptotic neuronal death [237]
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Its proven ability to stimulate collagen and elastin, accelerate skin repair, provide antioxidant protection, and enhance hydration positions it as a cornerstone of anti-aging and reparative formulations
It also activates the NO system and demonstrates broad cytoprotective properties
The mRNA and protein expression of the GSH biosynthesis pathway (GCLC, GCLM, GSS, and GSR) were downregulated in GCLC knockdown cells (Fig
Cancer Lett 407:139147
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